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    Advanced Cardiovascular Life Support Algorithms

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    Adult Bradycardia With a Pulse Algorithm

    Last reviewed October 19, 2026 · Based on ILCOR and AHA guidelines 2025-2030

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    ACLS Adult Bradycardia With a Pulse Algorithm flowchart

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    Adult Bradycardia With a Pulse Algorithm: Step by Step

    1. Assess the heart rate

      Determine whether the heart rate is appropriate for the patient’s condition. Bradyarrhythmia typically involves a heart rate <50/min.

    2. Is there cardiopulmonary compromise?

      Signs may include low blood pressure (hypotension), ischemic chest discomfort, signs of shock, acute change in mental status, and acute heart failure.

      • Yes: provide assessment and support
      • No: treat and monitor
    3. Compromise present: assessment and support

      Then check whether bradycardia persists with cardiopulmonary compromise.

      • Keep the airway open and administer oxygen
      • Support breathing with positive-pressure ventilation as needed
      • Connect the patient to a cardiorespiratory monitor
      • Continue monitoring the pulse
    4. No compromise: treat and monitor

      • Identify and treat potential causes
      • Support airway, breathing, and circulation
      • Consider supplemental oxygen
      • Obtain a 12-lead ECG
      • Continue monitoring the patient
    5. Does bradycardia persist with cardiopulmonary compromise?

      • No: treat and monitor
      • Yes: administer atropine
    6. Administer atropine

      If atropine is ineffective, use transcutaneous pacing and/or a dopamine or epinephrine infusion.

    7. Next steps

      • Consider expert consultation
      • Assess the need for transvenous pacing

    Medication dosing

    • Atropine IV bolus: first dose 1 mg; repeat every 3-5 minutes; maximum total dose 3 mg
    • If atropine is ineffective, dopamine IV infusion: infuse 5-20 mcg/kg per minute; adjust to clinical response and reduce gradually when discontinuing
    • Or epinephrine IV infusion: infuse 2-10 mcg per minute; adjust to clinical response

    Potential causes

    • Myocardial ischemia or infarction
    • Medication effects or toxicity, including beta-blockers, calcium channel blockers, and digoxin
    • Hypoxia: inadequate tissue oxygenation
    • Electrolyte disturbances, including hyperkalemia
    • Metabolic or endocrine disorders, such as acidosis or hypothyroidism
    • Hypothermia
    • Increased vagal tone
    • Structural heart disease or infection

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